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Regional hemodynamics after chronic nitric oxide inhibition in spontaneously hypertensive rats
(Elsevier Science Inc, New York, 2000)
Background: Inhibition of nitric oxide (NO) synthase by L-arginine analogs is associated with elevation of blood pressure in rats. Because endothelium-dependent vasomotion in different vascular beds is not homogenous, the ...
Consequences of angiotensin II type 1 receptor blockade on renal hemodynamic and morphology in adriamycin-induced nephropathy
(Lippincott Williams & Wilkins, Philadelphia, 2002)
Mean arterial pressure has not normalized after losartan infusion in experimental acute renal failure associated with NO deficiency
(Lippincott Williams & Wilkins, Philadelphia, 2002)
Biochemical consequences of acute no blokade and ET-1 infusion in Wistar rats
(Lippincott Williams & Wilkins, Philadelphia, 2003)
Experimental and systems biology studies of the molecular basis for the radioresistance of prostate carcinoma cells
(Springer, New York, 2008)
Molecular mechanisms for the gamma-ionizing radiation (IR) resistance of human prostate cancer cells, PC-3, are not quite clear. Since the low-LET-IR effects are primarily manifested by the generation of reactive oxygen ...
Hydroxyurea nitrosylates and activates soluble guanylyl cyclase in human erythroid cells
(Elsevier Science Inc, New York, 2007)
Effects of Nitric Oxide on Red Blood Cell Development and Phenotype
(Current Topics in Developmental Biology, 2008)
Nitric oxide (NO) is a diffusible free radical generated primarily by NO synthases (NOS), isoenzymes that convert the l-arginine and molecular oxygen to citrulline and NO in cells. Endothelial cells as well as macrophages, ...
Muscle strength testing: evaluation of tests of explosive force production
(Springer, New York, 2004)
The purpose of the study was to evaluate four tests of explosive force production (EFP). Specifically, the main aims of the study were to assess the reliability of different EFP tests, to examine their relationship with ...
Bosentan reduces blood pressure and improves kidney function and morphology in mild NO deficiency assotiated with experimental acute renal failure
(Lippincott Williams & Wilkins, Philadelphia, 2008)
Vit C decreases blood pressure, improves renal haemodynamics and reduces tubular dilatation in shr with postischemic acute renal failure (ARF)
(Lippincott Williams & Wilkins, Philadelphia, 2008)